Improvement by nefiracetam of beta-amyloid-(1-42)-induced learning and memory impairments in rats.

UI - 99158450

PMID- 10051141

DA - 19990503

DCOM- 19990503

LR - 20011128

IS - 0007-1188

VI - 126

IP - 1

DP - 1999 Jan

TI - Improvement by nefiracetam of beta-amyloid-(1-42)-induced learning and memory impairments in rats.

PG - 235-44 AB - 1. We have previously demonstrated that continuous i.c.v. infusion of amyloid beta-peptide (A beta), the major constituent of senile plaques in the brains of patients with Alzheimer's disease, results in learning and memory deficits in rats. 2. In the present study, we investigated the effects of nefiracetam [N-(2,6-dimethylphenyl)-2-(2-oxo-1-pyrrolidinyl) acetamide, DM-9384] on A beta-(1-42)-induced learning and memory deficits in rats. 3. In the A beta-(1-42)-infused rats, spontaneous alternation behaviour in a Y-maze task, spatial reference and working memory in a water maze task, and retention of passive avoidance learning were significantly impaired as compared with A beta-(40-1)-infused control rats. 4. Nefiracetam, at a dose range of 1-10 mg kg(-1), improved learning and memory deficits in the A beta-(1-42)-infused rats when it was administered p.o. 1 h before the behavioural tests. 5. Nefiracetam at a dose of 3 mg kg(-1) p.o. increased the activity of choline acetyltransferase in the hippocampus of A beta-(1-42)-infused rats. 6. Nefiracetam increased dopamine turnover in the cerebral cortex and striatum of A beta-(1-42)-infused rats, but failed to affect the noradrenaline, serotonin and 5-hydroxyindoleacetic acid content. 7. These results suggest that nefiracetam may be useful for the treatment of patients with Alzheimer's disease.

AD - Department of Neuropsychopharmacology and Hospital Pharmacy, Nagoya

University School of Medicine, Japan.

FAU - Yamada, K

AU - Yamada K

FAU - Tanaka, T

AU - Tanaka T

FAU - Mamiya, T

AU - Mamiya T

FAU - Shiotani, T

AU - Shiotani T

FAU - Kameyama, T

AU - Kameyama T

FAU - Nabeshima, T

AU - Nabeshima T

LA - eng

PT - Journal Article

CY - ENGLAND

TA - Br J Pharmacol

JID - 7502536

RN - 0 (Amyloid beta-Protein)

RN - 0 (Biogenic Monoamines)

RN - 0 (Peptide Fragments)

RN - 0 (Psychotropic Drugs)

RN - 0 (Pyrrolidinones)

RN - 102-32-9 (3,4-Dihydroxyphenylacetic Acid)

RN - 306-08-1 (Homovanillic Acid)

RN - 51-61-6 (Dopamine)

RN - 77191-36-7 (nefiracetam)

RN - EC 2.3.1.6 (Choline O-Acetyltransferase)

SB - IM

MH - 3,4-Dihydroxyphenylacetic Acid/metabolism

MH - Amyloid beta-Protein/*pharmacology

MH - Animal

MH - Avoidance Learning/drug effects

MH - Biogenic Monoamines/metabolism

MH - Choline O-Acetyltransferase/drug effects/metabolism

MH - Corpus Striatum/drug effects/enzymology

MH - Dopamine/metabolism

MH - Frontal Lobe/drug effects/enzymology

MH - Hippocampus/drug effects/enzymology

MH - Homovanillic Acid/metabolism

MH - Learning/drug effects

MH - Learning Disorders/chemically induced/*drug therapy

MH - Male

MH - Maze Learning/drug effects

MH - Memory Disorders/chemically induced/*drug therapy

MH - Motor Activity/drug effects

MH - Peptide Fragments/*pharmacology

MH - Psychotropic Drugs/pharmacology/*therapeutic use

MH - Pyrrolidinones/pharmacology/*therapeutic use

MH - Rats

MH - Rats, Wistar

MH - Support, Non-U.S. Gov't

EDAT- 1999/03/02

MHDA- 1999/03/02 00:01

PST - ppublish

SO - Br J Pharmacol 1999 Jan;126(1):235-44.


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